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Researchers continue to investigate whether the broad gene-regulatory actions attributed to GHK-Cu may support tissue architecture and cellular resilience through coordinated effects on collagen synthesis, antioxidant defence and extracellular matrix remodelling

The pathway analysis by KEGG on differentially expressed proteins suggested that the significantly affected pathways were steroid hormone biosynthesis, propanoate metabolism, systemic lupus erythematosus, non-alcoholic fatty liver disease (NAFLD), fatty acid degradation, fatty acid metabolism, citrate cycle (TCA cycle), carbon metabolism, fat digestion and absorption, Parkinsons disease (PD), oxidative phosphorylation, ubiquinone and other terpenoid-quinone biosynthesis, alcoholism (Fig

doi: 10.1007/BF01956868

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